Molecular neurobiology for the clinician by Dennis S Charney

By Dennis S Charney

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Macrocephaly, language level, general level of intellectual functioning, blood (platelet) serotonin levels, degree of social and communication impairment, presence of seizure disorder, gender of proband, dysmorphology, and severity or specific nature of restricted and repetitive behaviors are phenotypes of particular interest. An alternative approach might be to map loci for some of the traits that cluster in families of a probands with autism. 05%, with more affected males. GTS, other 22 MOLECULAR NEUROBIOLOGY FOR THE CLINICIAN tic disorders, and symptoms or full expression of childhoodonset OCD are associated both within individuals and within families.

1992). Higher recurrence rates are identified when subtypes of ADHD are considered, particularly ADHD with comorbid conduct disorder or ADHD that persists into adolescence (Faraone et al. 2000). Using subtypes with a higher genetic loading may increase power to detect association or linkage (J. Holmes et al. 2002). The genetic studies in ADHD provide a representative picture of genetic approaches in child and adolescent psychiatry. The two statistical approaches used in child and adolescent psychiatric disorders are linkage and association studies.

2002). It is important to note that the development of the nervous system is highly dependent on molecular recognition to provide appropriate signals for development. Knockout of major histocompatibility class I function in mice led to non-immune-mediated facilitation of long-term potentiation and absence of long-term depression (Huh et al. 2000). Molecular methodology is now being applied in postmortem studies of autism. These studies have tremendous potential, but initial findings may implicate a different distribution of neuronal types, numbers, and size, rather than implicating particular genes or protein systems in the pathophysiology of disease.

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Molecular neurobiology for the clinician by Dennis S Charney
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